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GOTCHAđŸ’„â€Œ Have we found the source of the clots???

Based on the available research, it is reasonable to conclude that both HIV (through gp120) and SARS-CoV-2 contribute to widespread cellular senescence in the brain, which indeed leads to increased levels of beta-secretase (BACE1). This, in turn, does open an additional route for amyloid formation. https://www.aging-us.com/article/101362/text In conclusion, the evidence strongly supports that both HIV […]

GOTCHAđŸ’„â€Œ Have we found the source of the clots??? Read More »

Could mRNA vaccine bacteria DNA contamination lead to cancers? New rumours of evidence!

From Dr. Mikolaj Raszek Approximately a year ago one of the most condemning information about mRNA vaccines came out indicating that the vaccines are contaminated with bacterial DNA (plasmid DNA) that was used for the production of mRNA for vaccines. At the time, two primary sources of this information came from the laboratories of Dr.

Could mRNA vaccine bacteria DNA contamination lead to cancers? New rumours of evidence! Read More »

Persistence of spike protein at the skull-meninges-brain axis may contribute to the neurological sequelae of COVID-19

SARS-CoV-2 infection is associated with long-lasting neurological symptoms, although the underlying mechanisms remain unclear. Using optical clearing and imaging, we observed the accumulation of SARS-CoV-2 spike protein in the skull-meninges-brain axis of human COVID-19 patients, persisting long after viral clearance. Further, biomarkers of neurodegeneration were elevated in the cerebrospinal fluid from long COVID patients, and

Persistence of spike protein at the skull-meninges-brain axis may contribute to the neurological sequelae of COVID-19 Read More »

COVID-19 immunopathology: From acute diseases to chronic sequelae

Innate cell‐mediated immunopathology in COVID‐19: Upon viral entry, there is a cascade of events that leads to inflammation, vascular damage, and blot. Tissue‐resident alveolar macrophages (AMs) and interstitial macrophages are among the first responders to SARS‐CoV‐2, which secret inflammatory cytokines including TNF, IL‐6, IL‐1ÎČ, and CCL2 that in addition to building up local inflammation but

COVID-19 immunopathology: From acute diseases to chronic sequelae Read More »

SARS-CoV-2 infection triggers several mechanisms that can accelerate aging

SARS-CoV-2 infection triggers several mechanisms that can accelerate aging: DNA Damage and Impaired Repair https://www.nature.com/articles/s41556-023-01097-w Cellular Senescence https://www.nature.com/articles/s41556-023-01097-w https://www.nature.com/articles/s43587-022-00170-7 Telomere Attrition https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2024.1399676/full Epigenetic Age Acceleration https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2024.1399676/full Immune System Dysregulation https://immunityageing.biomedcentral.com/articles/10.1186/s12979-023-00406-z Metabolic Disruption https://www.nature.com/articles/s41598-024-70612-2

SARS-CoV-2 infection triggers several mechanisms that can accelerate aging Read More »

Reevaluating Spikeopathy: A Deep Dive into the Molecular Pathogenicity and Risks of Gene-Based Technologies”

  This first paper explores peer-reviewed data counter to the ‘safe and effective’ narrative attached to these new technologies. Spike protein pathogenicity, termed ‘spikeopathy’, whether from the SARS-CoV-2 virus or produced by vaccine gene codes, akin to a ‘synthetic virus’, is increasingly understood in terms of molecular biology and pathophysiology. Pharmacokinetic transfection through body tissues

Reevaluating Spikeopathy: A Deep Dive into the Molecular Pathogenicity and Risks of Gene-Based Technologies” Read More »

Altered ACE2 and interferon landscape in the COVID-19 microenvironment correlate with the anti-PD-1 response in solid tumors

Ground zero for the formation process of calamari clots is a loss of the glycocalyx layer, which gets “replaced” layer by layer with the amyloid/hydrogel-like aggregation that coats the endothelial layer in the blood vessels. The microclots contribute and are enclosed in the less dense amyloid matrix. While reading this paper, I realized that some

Altered ACE2 and interferon landscape in the COVID-19 microenvironment correlate with the anti-PD-1 response in solid tumors Read More »

LILRB1 & LILRB2: Emerging Biomarkers for Long COVID’s Pro-Inflammatory State 🌟

The most frequently downregulated genes in significantly altered functional categories were two leukocyte immunoglobulin like receptors LILRB1 (FDR p = 0.005) and LILRB2 (FDR p = 0.027). PCA analysis demonstrated that LILRB1 and LILRB2 expression ‌discriminated all of the Long COVID samples â€ŒđŸ’„from COVID recovered samples. Discussion: Downregulation of these inhibitory receptors similarly indicates a

LILRB1 & LILRB2: Emerging Biomarkers for Long COVID’s Pro-Inflammatory State 🌟 Read More »

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