September 2024

COVID-19’s Double Threat: Arterial and Red Blood Cell Stiffness

This video highlights the critical issue of arterial stiffness following even mild COVID-19 infections, which is compounded by stiff red blood cells losing their flexibility. This combination significantly increases the risk of thrombosis, as stiff arteries and red blood cells together prevent proper blood flow through small vessels, leading to severe complications in COVID-19 patients.

COVID-19’s Double Threat: Arterial and Red Blood Cell Stiffness Read More »

Toxic Spike Protein Fragments: Micro Clotting in COVID-19

This video discusses how degraded spike proteins, broken down by neutrophil elastase, become highly toxic and thrombogenic, contributing to clot formation and micro clotting in COVID-19 patients. It highlights how persistent spike proteins in endothelial cells lead to vascular damage, and how targeting these proteins, rather than using blood thinners, may be a more effective

Toxic Spike Protein Fragments: Micro Clotting in COVID-19 Read More »

Microvascular Clotting and Circulatory Injury in COVID-19

This video explains the presence of microvascular clotting in COVID-19 patients, which can block tiny blood vessels and lead to symptoms like skin burning and brain fog. The discussion highlights the dangers of treating platelet-deficient patients with anticoagulants, which can cause bleeding, and emphasizes how viral persistence, immune dysregulation, and vascular damage are central to

Microvascular Clotting and Circulatory Injury in COVID-19 Read More »

COVID and Coagulation: A Personal Story of Recurring Issues

This video discusses a personal experience with recurring coagulation issues following COVID-19 infections, including elevated D-dimer levels and microvascular symptoms like double vision. The speaker reflects on the potential genetic predisposition to coagulation problems within their family, highlighting the importance of monitoring these symptoms post-infection.

COVID and Coagulation: A Personal Story of Recurring Issues Read More »

Myocarditis and Heart Damage: The Role of SARS-CoV-2 and Vaccines

This video addresses the dual threat of myocarditis and myocardial necrosis, which can occur from both SARS-CoV-2 infection and vaccination. The speaker emphasizes the importance of protecting the heart by lowering troponin levels, which can indicate heart damage. Additionally, they discuss how the spike protein contributes to heart damage, including the fusion of cardiomyocytes, exacerbating

Myocarditis and Heart Damage: The Role of SARS-CoV-2 and Vaccines Read More »

Targeting Senescent Cells and Spike Protein Debris

This video explains how SARS-CoV-2’s spike protein triggers cellular senescence, turning cells into non-functional states. The speaker describes how viral debris, including spike protein and syncytia, can be selectively removed through a slow, careful process, allowing the body to regenerate. The final step involves using binders and chelators to remove the liberated virus and debris

Targeting Senescent Cells and Spike Protein Debris Read More »

Senescent Cells and Syncytia: The Lasting Impact of SARS-CoV-2

This video explains how SARS-CoV-2 can turn cells into “zombie cells” that enter a senescent state, unable to die, repair, or replicate. These cells fuse into syncytia, creating conglomerates that harbor spike proteins and the virus, leading to impaired oxygen exchange in the lungs. This phenomenon was found in 67% of lung pathology cases, contributing

Senescent Cells and Syncytia: The Lasting Impact of SARS-CoV-2 Read More »

Spike Protein Impact: Widespread Damage from Virus and Vaccine

This video discusses the widespread effects of the SARS-CoV-2 virus and spike protein on the body, including how the vaccine’s spike protein production may cause more persistent and diverse organ damage. The conversation highlights the variability in COVID-19 symptoms and outcomes based on immunity and how the vaccine’s bio-distribution may lead to longer-lasting autoimmune responses

Spike Protein Impact: Widespread Damage from Virus and Vaccine Read More »

Spike Protein Factories: Virus vs. Vaccine Entry Mechanisms

This video explains the entry mechanisms of SARS-CoV-2 and vaccines, comparing how the virus relies on receptors to enter cells, while vaccine-delivered lipid nanoparticles forcefully fuse with any cell, turning it into a spike protein factory. It highlights the concerning exosome shedding of spike proteins, which can spread to other cells like naturally occurring lipid

Spike Protein Factories: Virus vs. Vaccine Entry Mechanisms Read More »

Spike Proteins Persisting in Brain and Olfactory System

This video highlights groundbreaking research showing the persistence of spike proteins and SARS-CoV-2 long after infection or vaccination. One study found spike proteins in vaccinated patients 187 days later, suggesting ongoing production from transfected cells, while other findings show viral presence in COVID-19 patients up to 654 days after recovery, particularly in the brain and

Spike Proteins Persisting in Brain and Olfactory System Read More »

Scroll to Top

Team Member 1

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo. Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo. Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo. Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo. Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo. Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.