Immuno Senescense

This video examines how dysfunctional and inflammatory immune cells, including senescent monocytes and macrophages, cause widespread tissue damage. It discusses the potential of senolytics in reversing immune senescence and the broader impact on other immune cells affected by spike protein persistence.

This video explores the widespread presence of spike proteins in the body and their role in long COVID. It highlights research on how compounds like quercetin and luteolin impact recovery, along with the slow, damaging effects of SARS-CoV-2 on the body.

This video explains how SARS-CoV-2’s spike protein triggers cellular senescence, turning cells into non-functional states. The speaker describes how viral debris, including spike protein and syncytia, can be selectively removed through a slow, careful process, allowing the body to regenerate. The final step involves using binders and chelators to remove the liberated virus and debris from the system. 

This video explains how SARS-CoV-2 can turn cells into “zombie cells” that enter a senescent state, unable to die, repair, or replicate. These cells fuse into syncytia, creating conglomerates that harbor spike proteins and the virus, leading to impaired oxygen exchange in the lungs. This phenomenon was found in 67% of lung pathology cases, contributing to poor gas exchange and long-term lung damage in COVID-19 patients. 

This video discusses the widespread effects of the SARS-CoV-2 virus and spike protein on the body, including how the vaccine’s spike protein production may cause more persistent and diverse organ damage. The conversation highlights the variability in COVID-19 symptoms and outcomes based on immunity and how the vaccine’s bio-distribution may lead to longer-lasting autoimmune responses compared to the virus itself. 

This video explains the entry mechanisms of SARS-CoV-2 and vaccines, comparing how the virus relies on receptors to enter cells, while vaccine-delivered lipid nanoparticles forcefully fuse with any cell, turning it into a spike protein factory. It highlights the concerning exosome shedding of spike proteins, which can spread to other cells like naturally occurring lipid nanoparticles. 

This video explains how spike proteins in infected cells cause irreversible damage, leading to the release of harmful cytokines like TNF-alpha and interleukin-6, which are prominent in long COVID. The speaker emphasizes the effectiveness of senolytics in selectively eliminating these damaged cells, with early tests showing a rapid release of spike proteins into the bloodstream after treatment.