Cardiovascular

This video explains how microvascular clotting and infected cells in COVID-19 patients cause reactive blood pressure and pulse fluctuations. It emphasizes the importance of addressing underlying vascular dysfunction to manage these symptoms effectively. 

This video highlights the critical issue of arterial stiffness following even mild COVID-19 infections, which is compounded by stiff red blood cells losing their flexibility. This combination significantly increases the risk of thrombosis, as stiff arteries and red blood cells together prevent proper blood flow through small vessels, leading to severe complications in COVID-19 patients.

This video discusses how degraded spike proteins, broken down by neutrophil elastase, become highly toxic and thrombogenic, contributing to clot formation and micro clotting in COVID-19 patients. It highlights how persistent spike proteins in endothelial cells lead to vascular damage, and how targeting these proteins, rather than using blood thinners, may be a more effective approach to reducing micro clotting.

This video explains the presence of microvascular clotting in COVID-19 patients, which can block tiny blood vessels and lead to symptoms like skin burning and brain fog. The discussion highlights the dangers of treating platelet-deficient patients with anticoagulants, which can cause bleeding, and emphasizes how viral persistence, immune dysregulation, and vascular damage are central to COVID-19 pathology.

This video discusses a personal experience with recurring coagulation issues following COVID-19 infections, including elevated D-dimer levels and microvascular symptoms like double vision. The speaker reflects on the potential genetic predisposition to coagulation problems within their family, highlighting the importance of monitoring these symptoms post-infection.

This video addresses the dual threat of myocarditis and myocardial necrosis, which can occur from both SARS-CoV-2 infection and vaccination. The speaker emphasizes the importance of protecting the heart by lowering troponin levels, which can indicate heart damage. Additionally, they discuss how the spike protein contributes to heart damage, including the fusion of cardiomyocytes, exacerbating the effects.